Unveiling the role of circRBBP7 in myoblast proliferation and differentiation: A novel regulator of muscle development

肌发生 生物 心肌细胞 细胞生物学 骨骼肌 再生(生物学) RNA干扰 PI3K/AKT/mTOR通路 细胞生长 调节器 基因 信号转导 遗传学 核糖核酸 解剖
作者
Yufeng Yang,Kongwei Huang,Hancai Jiang,Shuwan Wang,Xiaoxian Xu,Yang Liu,Qingyou Liu,Mingsong Wei,Zhipeng Li
出处
期刊:The FASEB Journal [Wiley]
卷期号:38 (14) 被引量:1
标识
DOI:10.1096/fj.202302599rr
摘要

Muscle development is a multistep process regulated by diverse gene networks, and circRNAs are considered novel regulators mediating myogenesis. Here, we systematically analyzed the role and underlying regulatory mechanisms of circRBBP7 in myoblast proliferation and differentiation. Results showed that circRBBP7 has a typical circular structure and encodes a 13 -kDa protein. By performing circRBBP7 overexpression and RNA interference, we found that the function of circRBBP7 was positively correlated with the proliferation and differentiation of myoblasts. Using RNA sequencing, we identified 1633 and 532 differentially expressed genes (DEGs) during myoblast proliferation or differentiation, respectively. The DEGs were found mainly enriched in cell cycle- and skeletal muscle development-related pathways, such as the MDM2/p53 and PI3K-Akt signaling pathways. Further co-IP and IF co-localization analysis revealed that VEGFR-1 is a target of circRBBP7 in myoblasts. qRT-PCR and WB analysis further confirmed the positive correlation between VEGFR-1 and circRBBP7. Moreover, we found that in vivo transfection of circRBBP7 into injured muscle tissues significantly promoted the regeneration and repair of myofibers in mice. Therefore, we speculate that circRBBP7 may affect the activity of MDM2 by targeting VEGFR-1, altering the expression of muscle development-related genes by mediating p53 degradation, and ultimately promoting myoblast development and muscle regeneration. This study provides essential evidence that circRBBP7 can serve as a potential target for myogenesis regulation and a reference for the application of circRBBP7 in cattle genetic breeding and muscle injury treatment.
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