失调
门静脉血栓形成
微生物群
医学
血栓形成
肝病
胃肠病学
阶段(地层学)
门脉高压
内科学
疾病
免疫学
肝硬化
生物
生物信息学
古生物学
作者
Rali R Aleksandrova,Lianne M. Nieuwenhuis,Naomi Karmi,Shuyan Zhang,J. Casper Swarte,Johannes R. Björk,Ranko Gaćeša,Hans Blokzijl,Margery A. Connelly,Rinse K. Weersma,Ton Lisman,Eleonora A. Festen,Vincent E. de Meijer,Coby Annema,Stephan J.L. Bakker,Stefan P. Berger,Hans Blokzijl,Frank A.J. A. Bodewes,Marieke T. de Boer,Kevin Damman
标识
DOI:10.1016/j.jtha.2024.12.036
摘要
We observed profound differences in gut microbiota between patients with ESLD and controls, but minimal differences between patients with ESLD with or without PVT. In our cohort, a gut-derived proinflammatory state was not associated with presence of PVT in patients with ESLD.
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