关贸总协定
胚胎干细胞
突变
诱导多能干细胞
清脆的
突变体
心脏发育
遗传学
生物
细胞生物学
癌症研究
基因
转录因子
作者
Lingqun Ye,Yu You,Zhen‐Ao Zhao,Dandan Zhao,Xuan Ni,Yong Wang,Xing Fang,Miao Yu,Yongming Wang,Jun‐Ming Tang,Ying Chen,Zhenya Shen,Wei Lei,Shijun Hu
摘要
In summary, our study revealed the molecular mechanism of the GATA4T280M mutation in ASD. Understanding the roles of the GATA4-FGF16 axis in iPSC-CMs will shed light on heart development and provide novel insights for the treatment of ASD and other CHD disorders.
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(2025-6-4)
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