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蛋白激酶B
PI3K/AKT/mTOR通路
LY294002型
细胞生物学
MAPK/ERK通路
癌症研究
未折叠蛋白反应
化学
信号转导
生物
作者
Dongmei Yan,Rongyang Dai,Chunyan Duan,Shaokun Chen,You‐Ping Liu,Chuan-ning Chen,Hong Li
出处
期刊：PubMed
[National Institutes of Health]
日期：2010-12-01
卷期号：18 (12): 909-14
被引量：1
标识
DOI：10.3760/cma.j.issn.1007-3418.2010.12.007
摘要
Endoplasmic reticulum stress-induced Akt activation is mediated through PI3K and the PI3K/Akt pathway inactivation is involved in increased ERK activity in human hepatocellular carcinoma cells. The cross-talk between the PI3K/Akt and MEK/ERK cascades plays an important role in endoplasmic reticulum stress-induced human hepatocellular carcinoma cell cycle arrest.
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