Neutrophil attachment via Mac-1 ( α M β 2 ; CD11b/CD18; CR3) integrins induces PAD4 deimination of profilin and histone H3

Neutrophil adhesion to endothelia, entry into tissues and chemotaxis constitute essential steps in the immune response to infections that drive inflammation. Neutrophils bind to other cells and migrate via adhesion receptors, notably the