Crush syndrome or traumatic rhabdomyolysis constitutes the systemic changes seen after crush injury. The \npressure causes necrosis of muscle, and during revascularisation diffusion of calcium, sodium and water into \nthe muscle cells is seen, together with loss of potassium, phosphate, lactic acid, myoglobin and creatinine \nkinase. Untreated these changes can lead to: hyperkalemia, acidosis, acute renal failure and hypovolemic \nshock. This case report describes a patient developing rhabdomyolysis following fracture of femur. It was \ndiagnosed and treated early during the course of anesthesia.