Impact of obesity progression or regression on the longitudinal assessment of fibrosing ILD

Abnormalities in lung mechanics (restriction) and pulmonary gas exchange (hypoxemia) may jointly conspire to elicit exertional dyspnea and decrease exercise tolerance in patients with fibrosing interstitial lung diseases ( f -ILD) [1]. Obesity (body mass index (BMI)≥30 kg·m−2), a prevalent co-morbidity of f -ILD [2], may negatively impact on "static" ( e.g. , total lung capacity (TLC)) and dynamic (forced vital capacity (FVC)) lung volumes relevant to dyspnea genesis [3]. Footnotes This manuscript has recently been accepted for publication in the European Respiratory Journal . It is published here in its accepted form prior to copyediting and typesetting by our production team. After these production processes are complete and the authors have approved the resulting proofs, the article will move to the latest issue of the ERJ online. Please open or download the PDF to view this article. Conflict of Interest: All authors have nothing to disclose.