A retinal detachment is not really a detachment of the complete
retina, but rather the separation of the neural component from the
pigment component. The neural retina can be pulled off by the
vitreoretinal traction or raised by exudation or by a tumour, but damage
most commonly occurs when a break in its surface permits liquefied
vitreous to pass from the vitreous cavity into the potential space that
exists between the two retinal layers.
A break in the retinal surface alone is not enough to give rise to
detachment. Fluid can only collect beneath the neuroretina if it is both
available and has access to retinal break. Such circumstances arise when
the vitreous gel collapses and separates the posterior vitreous cortex
from the retinal surface. Aetiologically, retinal breaks may result either
from some induced weakness in the retina itself or from some apparent
interplay between the retina and the vitreous.
CONCLUSION AND RECOMMENDATION
This study confirms that the incidence of RRD was found
to increase with age and in association with myopia, cataract surgery,
fellow eye RD, familial predisposition, YAG capsulotomy and trauma.
Hence the fellow eye should be properly examined to rule out any
predisposing peripheral degeneration and should be given prophylactic
therapy. Early identification of PCR during surgery with proper anterior
vitrectomy and cleanup of vitreous from wound is essential to prevent
vitreous traction and subsequent development of RRD.
This study clearly indicates that anatomical attachment
after external buckling procedure does not correlate with functional
success. Duration of macular detachment and extent and elevation of
detachment are the main predictors for the functional recovery. Hence
patient education regarding early symptoms, periodic detailed fundus
examination especially in myopes and in postoperative patients who has
increased risk in the first year after surgery should be emphasized. In
those cases prompt early management may improve the visual outcome.