MAPK/ERK通路
细胞生物学
发病机制
信号转导
p38丝裂原活化蛋白激酶
生物
免疫学
作者
Fei Zhang,Rui Feng,Meng Tong,Jin Chen,Song Zhang,Yan Xu,Jialin Meng,Cheng Yang,Meng Zhang,Chang Yin Liang
标识
DOI:10.1016/j.intimp.2025.114959
摘要
The results of the present study revealed that the MIF/CD74/CD44 axis played a critical role in Th17 cell differentiation via the p38 MAPK pathway in CNP. Thus, targeting the MIF/CD74/CD44/p38 MAPK pathway may offer a novel therapeutic strategy for treating chronic prostatitis.
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