厚壁菌
促炎细胞因子
失调
药理学
肠道菌群
炎症体
生物
免疫学
受体
生物化学
炎症
16S核糖体RNA
基因
作者
Xiaobo Wei,Ma Ni,Wen‐Bin Yang,Jinhu Tian,Huiyan Liu,Haitian Fang
标识
DOI:10.1002/mnfr.202300643
摘要
PERJ probably inhibits the development of UC by suppressing the NLRP3 and MAPKs signaling pathways and regulating gut microbiota homeostasis, and can be considered as a potential resource for preventing UC.
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