神经炎症
创伤性脑损伤
间充质干细胞
小胶质细胞
肿瘤坏死因子α
脂肪组织
生物
神经干细胞
病理
医学
免疫学
内科学
炎症
内分泌学
干细胞
细胞生物学
精神科
作者
Chao Xu,Yunfeng Diao,Jing Wang,Jun Liang,Hai‐Huan Xu,Mingliang Zhao,Bin Zheng,Zuo Luan,Jingjing Wang,Xiping Yang,Meng‐Guang Wei,Jinghao Duan,Keqiang Wang,Chong Chen,Feng Chen,Dong Ming,Sai Zhang,Hongtao Sun,Xiaohong Li
出处
期刊:Stem Cells and Development
[Mary Ann Liebert, Inc.]
日期:2019-12-13
卷期号:29 (4): 222-234
被引量:49
标识
DOI:10.1089/scd.2019.0173
摘要
The secretome of mesenchymal stem cell (MSC) offers a series of immunoregulatory properties and is regarded as an effective method of mitigating secondary neuroinflammation induced by traumatic brain injury (TBI). The secretome of adipose-derived MSCs (ASC-ST) was collected under hypoxia conditions. Proteomics data were analyzed by liquid chromatography–tandem mass spectrometry (LC-MS/MS), and concentrations of major components were tested. After the TBI caused by an electric cortical contusion impactor, rats were injected ASC-ST through caudal veins for 7 days. The neurological functional prognosis of TBI rats was significantly improved, and the vasogenic edema of brain tissues that was measured 14 days after TBI was relieved by ASC-ST, corresponding to brain water content levels. ASC-ST ameliorated TBI-induced neuroinflammatory environments that caused the edema, the apoptosis of the neural cells, and the nerve fiber damage by increasing the number of M2 phenotypes present while reducing the number of M1 phenotype microglia present. Furthermore, interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-α) levels were reduced, whereas transforming growth factor-beta (TGF-β) and tumor necrosis factor-stimulated gene 6 protein (TSG-6) levels were increased after secretome treatment. Altogether, ASC-ST is capable of improving neural functioning by modulating TBI-induced neuroinflammation and its related secondary insults. ASC-ST may be one of the most promising candidates for regulating the secondary inflammatory reactions of central nervous systems for clinical use.
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