Caffeic acid attenuates hydrogen peroxide-induced inflammation in porcine small intestinal epithelial cells by inhibiting nuclear factor kappa-B -mediated NOD-like receptor thermal protein domain associated protein 3 inflammasome activation

-induced intestinal epithelial cell injury, and the mechanism may be related to the inhibition of NF-κB-mediated NLRP3 inflammasome activation. These findings were expected to provide a theoretical basis for more reasonable and effective application of CA, and provide new ideas for nutritional regulation measures to alleviate intestinal inflammation in piglets.