Esculentoside a attenuates airway inflammation in asthma by regulating mitochondrial apoptosis via the JAK2/STAT3 pathway

: EsA ameliorates OVA-induced airway inflammation in mice, likely by suppressing the JAK2/STAT3 signaling pathway and attenuating mitochondrial-dependent apoptosis. These findings suggest that EsA holds therapeutic potential as a novel anti-asthmatic agent targeting inflammatory and mitochondrial pathways.