MPTP公司
黑质
多巴胺能
神经保护
刺激
神经科学
神经调节
化学
脑深部刺激
脂质过氧化
药理学
帕金森病
下调和上调
医学
丘脑底核
运动前神经元活动
星形细胞增多症
生物
细胞生物学
雷公藤甲素
多巴胺能途径
多巴胺
内分泌学
作者
Lu Sun,Haocheng Qin,Bao Zhou,Yixi Hao,Z. F. Ding,He Zhong,Ya Zheng,Yulian Zhu
标识
DOI:10.1186/s12967-026-08856-x
摘要
Parkinson’s disease (PD) is a neurodegenerative disorder characterized by the progressive loss of dopaminergic neurons in the substantia nigra (SN). Currently, there is a lack of effective therapies to slow disease progression. Temporal interference (TI) stimulation, as a novel non‑invasive neuromodulation technique, can generate a focused electric field in deep brain regions; however, whether it can effectively act on the SN and influence PD-related pathological processes remains unclear. To investigate the neuroprotective effects of TI stimulation on dopaminergic neurons in the SN of MPTP mice and explore the potential mechanisms involved. After establishing a subacute MPTP model, TI stimulation with a 10 Hz beat frequency was applied to the SN of mice for five consecutive days. Therapeutic outcomes and molecular mechanisms were evaluated using behavioral tests, immunohistochemistry, Western blot, transcriptome sequencing, untargeted lipidomics, and primary neuronal culture experiments. TI stimulation significantly improved motor function in MPTP mice, increased the number of TH‑positive neurons in the SN, and attenuated glial activation and neuroinflammation. Integrated transcriptomic and lipidomic analyses revealed that TI intervention downregulated PLA2G3. Experimentally, TI reduced the expression of ACSL4, upregulated GPX4, decreased MDA levels, elevated reduced GSH levels, and suppressed lipid peroxidation. In vitro experiments further confirmed that TI reduced MPP⁺‑induced primary neuronal death and lipid peroxidation accumulation. TI stimulation protects nigral dopaminergic neurons by inhibiting lipid peroxidation, ameliorates motor deficits and pathological features in PD mice, and provides a novel strategy for non‑invasive neuromodulation therapy in PD. Not applicable.
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