Prospective Evaluation of Intraorbital Soft Tissue Atrophy after Posttraumatic Bone Reconstruction: A Risk Factor for Enophthalmos

眼球内陷 软组织 轨道(动力学) 医学 眼眶骨折 萎缩 面部外伤 放射科 外科 病理 复视 工程类 航空航天工程
作者
Cristian Dinu,Tiberiu Tamaș,Gabriela Agrigoroaei,Sebastian Stoia,Horia Opriș,Simion Bran,Gabriel Armencea,Avram Manea
出处
期刊:Journal of Personalized Medicine [Multidisciplinary Digital Publishing Institute]
卷期号:12 (8): 1210-1210 被引量:2
标识
DOI:10.3390/jpm12081210
摘要

Orbital fractures are a common finding in facial trauma, and serious complications may arise when orbital reconstruction is not performed properly. The virtual planning can be used to print stereolithographic models or to manufacture patient-specific titanium orbital implants (PSIs) through the process of selective laser melting. This method is currently considered the most accurate technique for orbital reconstruction. Even with the most accurate techniques of bone reconstruction, there are still situations where enophthalmos is present postoperatively, and it may be produced by intraorbital soft tissue atrophy. The aim of this paper was to evaluate the orbital soft tissue after posttraumatic reconstruction of the orbital walls' fractures. Ten patients diagnosed and treated for unilateral orbital fractures were included in this prospective study. A postoperative CT scan of the head region with thin slices (0.6 mm) and soft and bone tissue windows was performed after at least 6 months. After data processing, the STL files were exported, and the bony volume, intraorbital fat tissue volume, and the muscular tissue volume were measured. The volumes of the reconstructed orbit tissues were compared with the volumes of the healthy orbit tissues for each patient. Our findings conclude that a higher or a lower grade of fat and muscular tissue loss is present in all cases of reconstructed orbital fractures. This can stand as a guide for primary or secondary soft tissue augmentation in orbital reconstruction.
最长约 10秒,即可获得该文献文件

科研通智能强力驱动
Strongly Powered by AbleSci AI
科研通是完全免费的文献互助平台,具备全网最快的应助速度,最高的求助完成率。 对每一个文献求助,科研通都将尽心尽力,给求助人一个满意的交代。
实时播报
Makubes发布了新的文献求助10
刚刚
MXL发布了新的文献求助10
1秒前
傅英俊完成签到,获得积分10
1秒前
dayan完成签到 ,获得积分10
1秒前
1秒前
秋风的应助被潇洒的诗桃采纳,获得10
2秒前
CZX完成签到,获得积分10
2秒前
Ashley发布了新的文献求助20
2秒前
洁净丹云发布了新的文献求助10
2秒前
3秒前
天天SCI发布了新的文献求助20
3秒前
3秒前
坚强土豆发布了新的文献求助10
4秒前
4秒前
NexusExplorer的应助被科研通管家采纳,获得10
4秒前
852的应助被科研通管家采纳,获得10
4秒前
隐形曼青的应助被科研通管家采纳,获得10
4秒前
乐乐的应助被科研通管家采纳,获得10
4秒前
华仔的应助被科研通管家采纳,获得10
4秒前
4秒前
852的应助被科研通管家采纳,获得10
4秒前
4秒前
搜集达人的应助被科研通管家采纳,获得10
5秒前
5秒前
桐桐的应助被科研通管家采纳,获得10
5秒前
ding的应助被科研通管家采纳,获得10
5秒前
yipmyonphu的应助被泥豪泥嚎采纳,获得10
5秒前
molihuakai的应助被科研通管家采纳,获得10
5秒前
研友_VZG7GZ的应助被科研通管家采纳,获得10
5秒前
Owen的应助被想人陪的雁凡采纳,获得10
5秒前
5秒前
可可的应助被科研通管家采纳,获得10
5秒前
5秒前
李健的应助被科研通管家采纳,获得10
6秒前
6秒前
赘婿的应助被科研通管家采纳,获得10
6秒前
hzl完成签到,获得积分10
6秒前
香蕉觅云的应助被科研通管家采纳,获得10
6秒前
6秒前
Dreamchaser发布了新的文献求助10
6秒前
高分求助中
(应助此贴封号)【重要!!请各用户(尤其是新用户)详细阅读】【科研通的精品贴汇总】 10000
CODESSA 2000
Rosenblum, Global Change Biology 800
Berberine regulates the TLR4 signaling pathway to suppress hypoxia-induced proliferation and migration of pulmonary arterial smooth muscle cells 520
Organizational Behavior 510
The Welfare Assembly Line: Public Servants in the Suffering City 500
Polymer-based Membranes for Separation and Recovery of Precious Metals 500
热门求助领域 (近24小时)
化学 材料科学 医学 生物 计算机科学 工程类 纳米技术 有机化学 化学工程 内科学 物理 生物化学 复合材料 催化作用 细胞生物学 人工智能 心理学 无机化学 基因 遗传学
热门帖子
关注 科研通微信公众号,转发送积分 7849114
求助须知:如何正确求助?哪些是违规求助? 9368953
关于积分的说明 20665334
捐赠科研通 7446285
什么是DOI,文献DOI怎么找? 3342630
关于科研通互助平台的介绍 2486218
邀请新用户注册赠送积分活动 2365835