特发性肺纤维化
肺纤维化
肺病
纤维化
疾病
医学
病理
免疫学
肺
内科学
作者
Richard Albert,David A. Schwartz
标识
DOI:10.1056/nejmcibr1811639
摘要
Pathogenic findings in a mouse model of idiopathic pulmonary fibrosis confirm the pivotal role played by dysfunction of the type 2 alveolar (AT2) epithelial cells in causing the disease. The findings support the use of this model in future investigations.
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