氧化磷酸化
FYN公司
细胞外基质
降级(电信)
细胞生物学
氧化应激
化学
细胞外
生物化学
激酶
生物
计算机科学
原癌基因酪氨酸蛋白激酶Src
电信
作者
Yichao Yan,Jing Peng,Wei He,Yan Chen,Wei Ye,Zongzhi Yin,Yunxia Cao
出处
期刊:Life Sciences
[Elsevier BV]
日期:2025-08-30
卷期号:380: 123939-123939
被引量:1
标识
DOI:10.1016/j.lfs.2025.123939
摘要
Our results revealed that SGK1 could potentially slow down the progression of POP under OS by interacting with GSK-3β to promote the GSK-3β (Ser9)/Fyn/NRF2 pathway, which provides a potential therapeutic approach for treating POP.
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