衰老
癌症研究
恶性转化
生物
转录组
表型
癌细胞
癌症
细胞
基因签名
黑色素瘤
肿瘤进展
细胞生物学
基因
遗传学
基因表达
作者
Alberta Palazzo,Héctor Hernández‐Vargas,Delphine Goehrig,Jean‐Jacques Médard,David Vindrieux,Jean‐Michel Flaman,David Bernard
出处
期刊:Cancer Letters
[Elsevier BV]
日期:2022-08-01
卷期号:546: 215850-215850
被引量:14
标识
DOI:10.1016/j.canlet.2022.215850
摘要
Oncogenic stress-induced senescence initially inhibits tumor initiation by blocking proliferation and by attracting immune cells to clear potentially harmful cells. If these cells are not eliminated they may resume proliferation upon loss-of-tumor suppressors, and be at risk of transformation. During tumor formation, depending on the sequence of events of gain-of-oncogenes and/or loss-of-tumor suppressors, cancer cells may emerge from senescent cells. Here, we show that these transformed cells after senescence (TS) display more aggressive tumorigenic features, with a greater capacity to migrate and a higher resistance to anti-tumoral drugs than cells having undergone transformation without senescence. Bulk transcriptomic analysis and single cell RNA sequencing revealed a signature unique to TS cells. A score of this signature was then generated and a high score was correlated with decreased survival of patients with lung adenocarcinoma, head-neck squamous cell carcinoma, adrenocortical carcinoma, liver hepatocellular carcinoma, skin cutaneous melanoma and low-grade glioma. Together, these findings strongly support that cancer cells arising from senescent cells are more dangerous, and that a molecular signature of these cells may be of prognostic value for some human cancers. It also raises questions about modeling human tumors, using cells or mice, without regards to the sequence of events leading to transformation.
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