基因敲除
癌症研究
生物
癌基因
细胞生长
细胞凋亡
肺癌
分子生物学
基因
遗传学
医学
细胞周期
病理
作者
Qiming Shen,Zhe Xu,Guanghao Sun,Haoyou Wang,Lin Zhang
标识
DOI:10.1158/1541-7786.mcr-22-0231
摘要
Our findings revealed that TFAP4 activated IGF2BP1 and facilitated NSCLC progression by stabilizing TK1 expression via m6A modification, which offered new insights into the diagnosis and treatment of NSCLC.
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