神经炎症
莫里斯水上航行任务
血管性痴呆
神经保护
药理学
医学
血脑屏障
血管生成
免疫印迹
痴呆
海马体
炎症
免疫学
内科学
生物
中枢神经系统
疾病
基因
生物化学
作者
Shitang Ma,Jianxin Jia,Lie Wu,Kai Tian,Lu Wang,Li Hua,Jiayu Lv,Dewang Gao,Zhan-Jun Yang,Xia Guo
标识
DOI:10.3389/fphar.2025.1640272
摘要
Objective: validation. Methods: The network pharmacology was used to explore the mechanism of TFDM to improve VaD. A rat model of VaD was established using permanent bilateral common carotid artery occlusion (2VO). The Morris water maze test assessed spatial learning and memory capacities. Nissl staining was used to examine the neuronal damage. Western blot and Immunofluorescence analysis was employed to evaluate protein levels of factors associated with neuroinflammation, blood-brain barrier integrity, and angiogenesis. Results: The network pharmacology suggests TFDM may combat VaD through TNF-α/NF-κB p65 signaling pathways. TFDM treatment may attenuate memory deficits associated with 2VO and reduce neuronal damage. TFDM improved blood-brain barrier integrity and promoted angiogenesis by downregulating MMP-9 and upregulating ZO-1 and VEGFA. Moreover, TFDM exhibited anti-inflammatory properties by inhibiting TNF-α and NF-κB p65 production, thereby mitigating the neuroinflammatory response in VaD rats. Conclusion: TFDM demonstrated significant improvement in cognitive function in VaD rats. This improvement was attributed to the multifaceted effects, including the improvement of blood-brain barrier integrity, promotion of angiogenesis, and reduction of neuroinflammation. These findings suggest that TFDM may represent a promising therapeutic approach for VaD management.
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