中性粒细胞胞外陷阱
肿瘤微环境
癌症研究
血液学
癌变
免疫系统
细胞外
癌症
DNA损伤
生物
癌症治疗
细胞生物学
免疫学
炎症
免疫疗法
肿瘤细胞
肿瘤进展
化学
细胞外基质
先天免疫系统
转移
癌症治疗
癌细胞
医学
实体瘤
DNA
癌症免疫疗法
过渡(遗传学)
免疫
作者
Jingwen He,Zhaokai Zhou,Guangyang Cheng,H. Li,Jiaqi Tu,Zixuan Fan,Xiuting Qiu,Wenjie Chen,Yuehua Chen,Li Li,Chen Li,Zhengrui Li,L Chen,Qian Lü
标识
DOI:10.1186/s13045-026-01789-4
摘要
Neutrophil extracellular traps (NETs), the DNA-protein structures released by neutrophils within the tumor microenvironment (TME), play a role in cancer that extends far beyond their traditional antimicrobial function. Specifically, NETs exert multifaceted and context-dependent effects on tumor progression, metastasis, immune regulation, and therapeutic response, showing their robust activity in shaping the complex landscape of tumor biology. This review systematically elucidates how NETs promote tumorigenesis by inducing DNA damage and epithelial-mesenchymal transition (EMT), and drive metastatic spread through mechanisms such as trapping circulating tumor cells, remodeling the pre-metastatic microenvironment, and reactivating dormant cells. Concurrently, NETs shape an immunosuppressive TME by modulating T cells, NK cells, and macrophages, while mediating resistance to chemotherapy, radiotherapy, and immunotherapy. Based on these mechanisms, targeting NET formation (such as inhibiting PAD4 or NE) or degrading their structures (such as using DNase I) has emerged as a potential strategy to enhance the efficacy of existing therapies. Therefore, deepening the understanding of the multifunctional regulatory networks underlying NETs within the TME holds significant implications for developing novel precision cancer therapies.
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