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Study Cover Figure: C-X-C motif chemokine ligand 1 (CXCL1)induced neutrophil infiltration and reactive oxygen species (ROS) production activates ROS-sensitive kinases and their downstream stress kinases (p38 and JNK) and subsequently enhances apoptotic signaling involving caspase-3 (CASP3) cleavage.Hwang and colleagues determined that interleukin (IL)-22Fc alleviates CXCL1-induced NASH by attenuating