γ-氨基丁酸受体
神经科学
抑制性突触后电位
补品（生理学）
串扰
受体
旁分泌信号
全身麻醉药
生物
认知
γ-氨基丁酸
麻醉剂
化学
药理学
医学
内科学
物理
麻醉
光学
作者
Dian-Shi Wang,Ju Li,Arsène Pinguelo,Kirusanthy Kaneshwaran,Sean Christopher Haffey,Irene Lecker,Himaben Gohil,Michael B. Wheeler,Lilia Kaustov,Anthony Ariza,MeiFeng Yu,Allen Volchuk,Benjamin E. Steinberg,Neil M. Goldenberg,Beverley A. Orser
标识
DOI：10.1016/j.trsl.2023.11.007
摘要
General anesthetic drugs cause cognitive deficits that persist after the drugs have been eliminated. Astrocytes may contribute to such cognition-impairing effects through the release of one or more paracrine factors that increase a tonic inhibitory conductance generated by extrasynaptic γ-aminobutyric acid type A (GABA
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