脂质过氧化
脂质代谢
抗氧化剂
APX公司
化学
生物化学
谷胱甘肽
采后
膜脂
DPPH
脂质氧化
生物
膜
超氧化物歧化酶
酶
植物
作者
Meiling Li,Hetong Lin,Chao Wang,Yazhen Chen,Mengshi Lin,Yen‐Con Hung,Yuan Lin,Zhengqiu Fan,Hui Wang,Yihui Chen
标识
DOI:10.1016/j.foodchem.2024.139175
摘要
Postharvest harmful pathogenic infestation leads to rapid decay in longan fruit. Compared with P. longanae-infected longans, AEOW alleviated fruit disease severity and diminished the O2-. production rate and MDA content. It also increased APX, CAT, and SOD activities, delayed the decrease in the levels of GSH and AsA, as well as the reducing power and DPPH radical scavenging ability, which resulted in a decline in membrane lipid peroxidation in P. longanae-infected longans. Additionally, AEOW reduced LOX, lipase, PI-PLC, PC-PLC, and PLD activities, maintained higher levels of PC, PI, IUFA, USFAs, and U/S, while reducing levels of PA, DAG, SFAs, and CMP. These effects alleviated membrane lipid degradation and peroxidation in P. longanae-infected longans. Consequently, AEOW effectively maintained membrane integrity via improving antioxidant capacity and suppressing membrane lipid peroxidation. This comprehensive coordination of ROS and membrane lipid metabolisms improved fruit resistance and delayed disease development in longans.
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