Mdivi1 ameliorates mitochondrial dysfunction in non‐alcoholic steatohepatitis by inhibiting JNK/MFF signaling

Collectively, Mdivi1 protected against diet-induced NASH by restoring mitochondrial homeostasis and function, potentially through its inhibitory effect on the JNK/MFF pathway. Consequently, further investigation of Mdivi1 as a promising drug for NASH treatment is warranted.