感染性休克
白细胞介素2受体
医学
FOXP3型
休克(循环)
免疫学
流式细胞术
细胞凋亡
败血症
T细胞
内科学
免疫系统
生物
遗传学
作者
Fabienne Venet,Alexandre Pachot,Anne‐Lise Debard,Julien Bohé,Jacques Bienvenu,Alain Lepape,Guillaume Monneret
标识
DOI:10.1097/01.ccm.0000145999.42971.4b
摘要
The increased percentage of Treg after shock is due not to their proliferation but to a decrease in CD4+CD25- T lymphocyte number. We hypothesize that it might be due to a resistance of Treg to apoptosis processes occurring during septic shock.
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