骨愈合
血管生成
移植
骨髓
细胞生物学
干细胞
内皮干细胞
癌症研究
免疫学
生物
医学
解剖
内科学
体外
生物化学
作者
Yoshihito Suda,Akihiko Taguchi,Tomoyuki Matsumoto,Yuka Okinaka,Shinya Hayashi,Masanori Tsubosaka,Tomoyuki Kamenaga,Yuichi Kuroda,Naoki Nakano,Yuma Onoi,Shotaro Tachibana,Kensuke Wada,Akira Saito,Takuma Maeda,S. Araki,Kohei Motono,Ryosuke Kuroda
出处
期刊:Stem Cells
[Wiley]
日期:2025-01-23
卷期号:43 (4)
被引量:1
标识
DOI:10.1093/stmcls/sxae090
摘要
Abstract Aims Bone marrow mononuclear cells (BM-MNCs) are a rich source of hematopoietic stem cells that have been widely used in experimental therapies for patients with various diseases, including fractures. Activation of angiogenesis is believed to be one of the major modes of action of BM-MNCs; however, the essential mechanism by which BM-MNCs activate angiogenesis remains elusive. This study aimed to demonstrate that BM-MNCs promote bone healing by enhancing angiogenesis through direct cell-to-cell interactions via gap junctions, in addition to a previously reported method. Methods Using a murine fracture model, we aimed to elucidate the relationship between gap junction-mediated cell-to-cell interactions and enhanced fracture healing after BM-MNC transplantation. We evaluated the transfer of substances from BM-MNCs to vascular endothelial cells and osteoblasts in the tissues surrounding the fracture site and assessed the effects of BM-MNC transplantation on bone healing, angiogenesis, and osteogenesis. Results Bone marrow mononuclear cells transferred substances to vascular endothelial cells and osteoblasts in the tissues surrounding the fracture site. Moreover, BM-MNC transplantation promoted bone healing via gap junction-mediated cell-to-cell interactions, accelerating both angiogenesis and osteogenesis. Conclusions Our findings provide a novel understanding of fracture healing mechanisms and suggest that BM-MNC transplantation enhances bone healing through gap junction-mediated cell-to-cell interactions, contributing to the development of regenerative medicine strategies targeting bone repair.
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