三阴性乳腺癌
医学
PI3K/AKT/mTOR通路
乳腺癌
癌症研究
蛋白激酶B
内科学
癌症
肿瘤科
信号转导
细胞生物学
生物
作者
Jianjie Li,Xiangmei Zhang,Xueliang Liu,Xiangmin Ma,Yanfang Wang,Yunjiang Liu
标识
DOI:10.1080/14737140.2024.2394167
摘要
NFYA promotes TNBC cell stemness by upregulating JARID2 expression and regulating the PI3K/AKT signaling pathway, suggesting JARID2 as a potential target for innovating drugs that target TNBC stem cells.
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