生物
自噬
小RNA
心肌梗塞
焊剂(冶金)
细胞生物学
细胞凋亡
心脏病学
基因
遗传学
材料科学
医学
冶金
作者
Liang Chen,Dongyang Jiang,Wenxin Kou,Yawei Xu
标识
DOI:10.1016/j.mcp.2025.102037
摘要
These findings reveal that miR-130 is a pivotal regulator of myocardial injury in AMI, mediating its effects via the modulation of autophagy and ferroptosis pathways. Targeting miR-130 may therefore represent a promising therapeutic strategy for mitigating myocardial damage and improving cardiac function following AMI.
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