逮捕
缺血
再灌注损伤
细胞生物学
药理学
心脏缺血
心肌保护
G蛋白偶联受体
化学
信号
医学
信号转导
心脏病学
生物
作者
Yimei Wang,Jin Li,Ying Song,Mao Zhang,Dan Shan,Yu‐Li Liu,Meng Fang,Fengxiang Lv,Rui‐Ping Xiao,Yan Zhang
摘要
We define an upregulation of Arrb2 as a pathogenic factor in cardiac I/R injury, and also reveal a novel GPCR-independent mechanism of Arrb2-mediated cell death signalling in the heart.
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