The Mechanism of Action of NMN in Inhibiting NLRP3 Acetylation‐Mediated Granulosa Cell Pyroptosis to Improve Ovarian Reserve Function in Ionizing Radiation‐Exposed Mice

-dependent deacetylase, plays a role in mitigating the decline in ovarian function by promoting the deacetylation of NLRP3 at lysine 570, thereby inhibiting the activation of the NLRP3 inflammasome. This study provides valuable insights into the potential of NMN supplementation in mitigating the adverse effects of radiation on ovarian function.