Arabidopsis Golgi Anti‐Apoptotic Proteins Confer Drought Tolerance to Water Deficiency by Enhancing Degradation of the Aquaporin PIP2;7

细胞生物学 拟南芥 水通道蛋白 内质网 渗透性休克 高尔基体 未折叠蛋白反应 生物 下调和上调 突变体 化学 生物化学 基因
作者
Yan Zhou,Yu‐Ting He,Xi Chen,Junwei Zhao,Wei Zhang,Jing Tan,Xin Li,Xin Li,Shu‐Heng Zhao,Yi‐Qiu Ning,Yue Sun,Xiao‐Fang Li,Xiao‐Fang Li
出处
期刊:Plant Cell and Environment [Wiley]
卷期号:48 (8): 6402-6405 被引量:1
标识
DOI:10.1111/pce.15617
摘要

Water retention is one of the important factors for plants to survive under various stress conditions. In plants, the so-called plasma membrane intrinsic proteins (PIPs) are the main water channels that regulate the water status of plants. Membrane trafficking contributes to the functional regulation of major PIPs and is crucial for abiotic stress resilience. Arabidopsis Golgi anti-apoptotic proteins (GAAPs) play redundant function in resisting endoplasmic reticulum stress-induced cell death. However, much less is known about the connection between the cellular homeostasis response and the resistance to water deficiency. In this study, we analyzed the function of GAAPs under salt stress, osmotic stress and drought using single and multiple mutants of GAAP1 to GAAP4. GAAPs conferred salt resistance redundantly and GAAP4 played a major role in the resistance to water shortage. Aquaporin PIP2;7 (PIP3) was found interacting with GAAP1-4 by yeast two hybrid, cellular and co-immunoprecipitation assays. Genetic evidence suggests that PIP3 was essential for the function of GAAP4 against osmotic stress. GAAPs mutation(s) delayed the downregulation of PIP3 levels under osmotic stress. The internalization of PIP3 and plasma membrane was suppressed by GAAP4 mutation. So the positive function of GAAPs against water shortage stress might be partly due to its positive effect on membrane PIP3 cycling and turnover, thereby reducing cell water loss. The data also lay foundation for further studies on the connection of water retention regulation with cell fate decision.
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