Ascorbic acid treatment inhibits early wound healing in the fresh-cut potato by relegating jasmonic acid biosynthesis and signal transduction

with AsA. AsA treatment reduced jasmonic acid carboxyl methyltransferase and jasmonic acid-amido synthetase activities, StJMT and StJAR1 expressions, and methyl jasmonate and jasmonoyl-isoleucine contents. Moreover, AsA treatment upregulated StJAZ1 and downregulated StMYC2 expression. These findings suggested that AsA regulates wound healing in FCPs by suppressing JA biosynthesis and signal transduction.