小胶质细胞
疾病
生物
神经科学
阿尔茨海默病
认知
干扰素
免疫学
出处
期刊:Immunity
[Elsevier]
日期:2022-05-01
卷期号:55 (5): 821-823
标识
DOI:10.1016/j.immuni.2022.04.008
摘要
Although consistently implicated, the exact role of interferon (IFN) signaling in Alzheimer's disease remains largely unexplored. Roy et al. now demonstrate that type I IFNs may drive cognitive dysfunction through acting not only on microglia but, surprisingly, also on neurons.
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