生物
机制(生物学)
萧条(经济学)
骨重建
单胺类神经递质
信号转导
新陈代谢
生物信息学
神经科学
内分泌学
细胞生物学
血清素
遗传学
受体
哲学
宏观经济学
经济
认识论
作者
Haiming Gao,Chen-Yi Huang,Kaili Zhao,Xueyan Chen,Xuemei Zhang,Yaoge Deng,Zongchao Liu,Dayue Darrel Duan
标识
DOI:10.1089/dna.2019.5284
摘要
Increasing evidence indicates that depression affects bone metabolism to some extent, but the specific mechanisms are still unclear. Numerous studies have confirmed that a variety of signaling molecules are involved in depression's impact on fracture healing, including serum monoamine neurotransmitters, cytokines, inflammatory markers, growth factors, and metabolites. This article comprehensively discusses the effects of depression-associated signaling molecules on bone metabolism and their underlying mechanisms to provide a basis for early preventive intervention for delayed fracture healing in patients with depression.
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