Effect of curcumin on the non-alcoholic steatohepatitis via inhibiting the M1 polarization of macrophages

姜黄素 肿瘤坏死因子α 脂多糖 标记法 M2巨噬细胞 脂肪性肝炎 化学 脂肪肝 巨噬细胞 流式细胞术 细胞凋亡 医学 巨噬细胞极化 免疫学 炎症 药理学 内科学 生物 生物化学 体外 疾病
作者
Cong Tong,Hongfei Wu,Da Gu,Yanian Li,Yuhan Fan,Jiahui Zeng,Wei Ding
出处
期刊:Human & Experimental Toxicology [SAGE]
卷期号:40 (12_suppl): S310-S317 被引量:24
标识
DOI:10.1177/09603271211038741
摘要

Background Non-alcoholic steatohepatitis (NASH) is a global medical problem and macrophages’ activation is closely related to the pathogenesis of NASH. Curcumin is a polyphenol from turmeric with significant anti-inflammatory activity. Objective The objective of present study was to observe the effect of curcumin on macrophages’ activation and secretion of pro-inflammatory cytokines in NASH. Methods Hematoxylin and eosin and TUNEL staining were used to observe the hepatic function. RT-PCR was conducted to evaluate the hepatic mRNA expression of tumor necrosis factor-α (TNF-α) and interleukin-1β (IL-1β). Flow cytometry was adopted to detect the M1 polarization of macrophages. The RAW264.7 macrophage was pretreated with different doses of curcumin, and then lipopolysaccharide (LPS) and interferon-γ (IFN-γ) were given to activate the M1 macrophage. The activation ratio of M1 macrophage was observed by flow cytometry, and IL-1β and TNF-α expression was detected by RT-PCR and ELISA. Results After treatment with curcumin, the activities of alanine aminotransferase (ALT) and aspartate aminotransferase (AST), the mRNA expression of TNF-α and IL-1β, and M1 polarization of macrophages were significantly decreased. Hematoxylin and eosin and TUNEL staining showed that inflammation and apoptosis in the liver were improved. What is more, curcumin can effectively inhibit M1 macrophage activation induced by lipopolysaccharide and IFN-γ and reduce the secretion of IL-1β and TNF-α. Conclusion Curcumin can effectively improve NASH and reduce hepatic cell necrosis by inhibiting the M1 polarization of macrophages and the secretion of inflammatory factors.
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