粒体自噬
品脱1
神经保护
莫里斯水上航行任务
氧化应激
细胞生物学
线粒体
化学
海马结构
自噬
神经科学
生物
生物化学
细胞凋亡
作者
Fanrui Zhao,Chunlei Liu,Li Fang,Hongyan Lu,Ji Wang,Yawen Gao,Rosita Gabbianelli,Weihong Min
标识
DOI:10.1021/acs.jafc.0c07546
摘要
-treated HT-22 cells. These data suggested that YVLLPSPK improved learning and memory in scopolamine-induced cognitive-impaired mice through a mechanism associated with PINK1-mediated mitophagy via the NRF2/KEAP1/HO-1 pathway.
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