医学
乙型肝炎表面抗原
抗体
免疫学
养生
乙型肝炎
免疫系统
T淋巴细胞
病毒学
抗原
B细胞
抗体反应
T细胞
慢性肝炎
肝炎
Cd4 t细胞
还原(数学)
慢性病
细胞
乙型肝炎病毒
作者
Taiyu He,Min Chen,Maoying Liu,L Zhang,Huidan Sun,L Zhang,Aoyi Li,Weiqun Zeng,Ning Ling,Xiaofeng Shi,Hua He,Mingli Peng,Dachuan Cai,Peng Hu,Dazhi Zhang,Yinghua Lan,Hong Ren
出处
期刊:Gut
[BMJ]
日期:2025-12-24
卷期号:: gutjnl-2025
被引量:3
标识
DOI:10.1136/gutjnl-2025-336655
摘要
BACKGROUND: PD-1 blockade has emerged as a promising approach for functional cure of chronic hepatitis B (CHB). OBJECTIVE: This study aimed to evaluate the safety profile of anti-PD-1 antibody (αPD-1), as well as its impact on hepatitis B surface antigen (HBsAg) and immune responses in a larger cohort of CHB patients. DESIGN: In this prospective, open-label study, virally suppressed patients with CHB on nucleos(t)ide analogue (NA) were assigned to receive either 24-week NA monotherapy (n=62) or αPD-1 (half-dose sintilimab) add-on therapy (n=59), with both groups subsequently receiving NA monotherapy for an additional 12-week observation period. RESULTS: IU/mL, p<0.001) and higher HBsAg loss rates (6.1% vs 0%, p=0.166) than NA monotherapy at week 24. Notably, significant HBsAg decline and seroclearance exclusively occurred in the initial 12 weeks of αPD-1 treatment. HBsAg levels did not rebound at 12 weeks after discontinuation of αPD-1 therapy. After αPD-1 therapy, the numbers of HBsAg-specific, HBpol-specific, HBx-specific and HBeAg/HBcAg-specific IFN-γ spots all increased (p<0.05), while frequencies of HBsAg-specific B cells remained stable. Furthermore, ALT elevation and enhanced HBsAg-specific T-cell responses following αPD-1 therapy correlated with HBsAg decline (p<0.05). CONCLUSIONS: In virally suppressed CHB patients on NA therapy, 24-week half-dose sintilimab treatment demonstrated a favourable safety profile. This regimen can facilitate HBsAg reduction and even HBsAg loss, while concurrently enhancing HBV-specific T-cell responses. These findings support αPD-1 as a potential therapeutic alternative for CHB. TRIAL REGISTRATION NUMBER: NCT05769816.
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