Strategies for managing the destruction of calcar femorale

医学 运动医学 骨科手术 痹症科 止痛药 内科学 物理疗法 外科 麻醉学 股骨 病理
作者
Mei Jin,Lili Pang,Zhongchao Jiang
出处
期刊:BMC Musculoskeletal Disorders [BioMed Central]
卷期号:22 (1) 被引量:10
标识
DOI:10.1186/s12891-021-04324-3
摘要

Abstract Background The calcar femorale was identified long ago. However, our current understanding of the calcar is insufficient, and its related concepts are sometimes confused. The calcar femoral is an important anatomical structure of the proximal femur, and its function can be overlooked. In trauma, tumors, or other diseases, the calcar femorale can be destroyed or changed pathologically. As a result, the mechanical structure of the proximal femur becomes destroyed, causing pathological fractures. How to address the destruction of the calcar femorale or the damage to the calcar femorale is discussed in this article. Main text Destruction of the calcar femorale is accompanied by many conditions, including trauma, tumors, and other diseases. The types of hip fractures caused by trauma include femoral neck fractures and intertrochanteric fractures. Dynamic hip screws, proximal femoral nail anti-rotation, and multiple parallel cannulate pins can be used in different conditions. When metastatic and primary bone tumors involve the calcar femorale, endoprostheses are widely used. Other diseases, such as fibrous dysplasia and aneurysmal bone cyst are treated differently. Conclusions The calcar femorale can redistribute stresses and the destruction of the calcar femorale can lead to an increase in posterior medial stress. Many factors need to be considered when deciding whether to reconstruct the calcar femorale. Effective treatment strategies for managing the destruction of calcar femorale will need first establishing the precise mechanism of the destruction of the calcar and then designing therapies towards these mechanisms. Further investigation to the calcar needs to be carried out.
最长约 10秒,即可获得该文献文件

科研通智能强力驱动
Strongly Powered by AbleSci AI
科研通是完全免费的文献互助平台,具备全网最快的应助速度,最高的求助完成率。 对每一个文献求助,科研通都将尽心尽力,给求助人一个满意的交代。
实时播报
动漫大师发布了新的文献求助10
刚刚
莫莫发布了新的文献求助10
1秒前
xiaoxiao完成签到,获得积分10
1秒前
乌萨奇完成签到,获得积分10
1秒前
1秒前
ZZDXXX完成签到,获得积分10
1秒前
清久完成签到,获得积分10
2秒前
2秒前
2秒前
华仔应助科研通管家采纳,获得10
3秒前
niyl完成签到,获得积分10
3秒前
JamesPei应助科研通管家采纳,获得10
3秒前
SYLH应助科研通管家采纳,获得10
3秒前
NexusExplorer应助科研通管家采纳,获得10
3秒前
汉堡包应助科研通管家采纳,获得10
3秒前
小蘑菇应助科研通管家采纳,获得10
3秒前
桐桐应助科研通管家采纳,获得10
3秒前
科研通AI5应助科研通管家采纳,获得10
3秒前
酷波er应助科研通管家采纳,获得10
3秒前
SYLH应助科研通管家采纳,获得10
4秒前
SYLH应助科研通管家采纳,获得10
4秒前
SYLH应助科研通管家采纳,获得10
4秒前
SYLH应助科研通管家采纳,获得10
4秒前
乐乐应助科研通管家采纳,获得10
4秒前
Orange应助科研通管家采纳,获得10
4秒前
领导范儿应助科研通管家采纳,获得10
4秒前
科研通AI5应助科研通管家采纳,获得10
4秒前
4秒前
若狂完成签到,获得积分10
4秒前
最好完成签到 ,获得积分10
4秒前
小蘑菇应助科研通管家采纳,获得10
4秒前
小二郎应助开心妙旋采纳,获得10
4秒前
JinwenShi完成签到,获得积分10
4秒前
4秒前
6666666666完成签到,获得积分10
4秒前
大知闲闲完成签到,获得积分10
4秒前
快乐难敌发布了新的文献求助10
4秒前
搜集达人应助科研通管家采纳,获得30
5秒前
5秒前
5秒前
高分求助中
Mass producing individuality 600
Algorithmic Mathematics in Machine Learning 500
Разработка метода ускоренного контроля качества электрохромных устройств 500
Advances in Underwater Acoustics, Structural Acoustics, and Computational Methodologies 300
Diagnostic Pathology: Kidney Diseases 200
Advanced Micropipette Techniques for Cell Physiology 200
Encyclopedia of Ocean Sciences Third Edition 200
热门求助领域 (近24小时)
化学 材料科学 医学 生物 工程类 有机化学 物理 生物化学 纳米技术 计算机科学 化学工程 内科学 复合材料 物理化学 电极 遗传学 量子力学 基因 冶金 催化作用
热门帖子
关注 科研通微信公众号,转发送积分 3827809
求助须知:如何正确求助?哪些是违规求助? 3369942
关于积分的说明 10460280
捐赠科研通 3089785
什么是DOI,文献DOI怎么找? 1700055
邀请新用户注册赠送积分活动 817656
科研通“疑难数据库(出版商)”最低求助积分说明 770325