瘢痕疙瘩
地址1
炎症
癌症研究
伤口愈合
医学
化学
细胞因子
信号转导
病理
角质形成细胞
促炎细胞因子
免疫学
下调和上调
作者
Yabing Hu,X. H. Xie,Huayu Huang,Longshan Li,Yixiao Xiong,Yuyang Zeng,Dan Li,Ying Xia,Yi Duan,Yongchu Huang,Yong Zhang,Xuyun Zhang
摘要
Collectively, these findings reveal the molecular basis of collagen I-DDR1 signaling activating NFκB and STAT3 signaling and expose the role of ARF6-DDR1 axis in promoting keloid inflammation development, indicating that DDR1 and ARF6 may serve as innovative therapeutic targets in keloid.
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(2025-6-4)
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