微泡
细胞生物学
再生（生物学）
活性氧
NADPH氧化酶
炎症
细胞外
轴突
小RNA
化学
生物
免疫学
生物化学
基因
作者
Eva‐Maria Krämer‐Albers
标识
DOI：10.1038/s41556-018-0048-9
摘要
Inflammation and reactive oxygen species (ROS) production after central nervous system injury are thought to enhance tissue damage and hamper neuronal regeneration. Evidence now suggests that NADPH2 oxidase delivery from macrophages to injured neurons through extracellular vesicles, promotes ROS signalling and axon recovery.
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