Operando quantum sensing captures the nanoscale electrochemical evolution in batteries

oscillations upon DHX9 overexpression.Using a strictly cytoplasmatic DHX9 mutant, we determined that these effects are not dependent on its nuclear localization.On the other hand, DHX9 silencing induced the reverse effect on both SOCE and carbachol-induced calcium oscillations.Finally, we demonstrated that DHX9 regulates focal adhesion-associated Ca 2 signaling and cell migration.In summary, we identified DHX9 as a novel interactor of the SARAF-STIM1 complex and SOCE regulator that impacts cell migration.