神经炎症
丙二醛
认知功能衰退
超氧化物歧化酶
肠-脑轴
氧化应激
肠道菌群
海马结构
生物
内科学
内分泌学
医学
免疫学
炎症
痴呆
疾病
标识
DOI:10.1021/acs.jafc.3c03183
摘要
< 0.05) by promoting the MAPK signaling pathway while enriching the abundance of butyrate-producing bacteria and altering the functional profile of the microbial community. In conclusion, ARE and CY3D5G may attenuate age-related cognitive dysfunction by reducing neuroinflammation and regulating the gut-brain axis.
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