Discovery of dual-specificity tyrosine-phosphorylation-regulated kinase 1A (DYRK1A) inhibitors using an artificial intelligence model and their effects on tau and tubulin dynamics

DYRK1A型 磷酸化 蛋白酪氨酸激酶 酪氨酸 酪氨酸激酶 化学 对偶(语法数字) 激酶 细胞生物学 微管蛋白 酪氨酸磷酸化 生物化学 癌症研究 计算生物学 生物 信号转导 微管 艺术 文学类
作者
J J Chen,Huang‐Ju Tu,Tony Eight Lin,Zhaoxiang Peng,Yiwen Wu,Shih‐Chung Yen,Tzu-Ying Sung,Jui‐Hua Hsieh,Hsueh‐Yun Lee,Shiow‐Lin Pan,Wei‐Chun HuangFu,Kai‐Cheng Hsu
出处
期刊:Biomedicine & Pharmacotherapy [Elsevier BV]
卷期号:181: 117688-117688 被引量:2
标识
DOI:10.1016/j.biopha.2024.117688
摘要

The dual-specificity tyrosine-phosphorylation-regulated kinase 1 A (DYRK1A) presents a promising therapeutic target for neurological diseases. However, current inhibitors lack selectivity, which can lead to unexpected side effects and increase the difficulty of studying DYRK1A. Therefore, identifying selective inhibitors targeting DYRK1A is essential for reducing side effects and facilitating neurological disease research. This study aimed to discover DYRK1A inhibitors through a screening pipeline incorporating a deep neural network (DNN) model. Herein, we report an optimized model with an accuracy of 0.93 on a testing set. The pipeline was then performed to identify potential DYRK1A inhibitors from the National Cancer Institute (NCI) library. Four novel DYRK1A inhibitors were identified, and compounds NSC657702 and NSC31059 were noteworthy for their potent inhibition, with IC50 values of 50.9 and 39.5 nM, respectively. NSC31059 exhibited exceptional selectivity across 70 kinases. The compounds also significantly reduced DYRK1A-induced tau phosphorylation at key sites associated with the pathology of neurodegenerative diseases. Moreover, they promoted tubulin polymerization, suggesting a role in microtubule stabilization. Cytotoxicity assessments further confirmed the neuronal safety of the compounds. Together, the results demonstrated a promising screening pipeline and novel DYRK1A inhibitors as candidates for further optimization and development.
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