EZH2型
淋巴瘤
癌症研究
免疫学
激活剂(遗传学)
生物
医学
内科学
甲基化
遗传学
基因
受体
出处
期刊:Blood
[Elsevier BV]
日期:2016-08-18
卷期号:128 (7): 888-889
被引量:7
标识
DOI:10.1182/blood-2016-06-723916
摘要
In this issue of Blood , [Yan et al][1] report their findings in natural killer/T-cell lymphoma (NKTL) and present experimental evidence that EZH2 can act as a transcriptional activator, independent of its methyltransferase activity, when phosphorylated by JAK3. This may open up the prospect of new
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