纤维化
天狼星红
PI3K/AKT/mTOR通路
蛋白激酶B
上皮-间质转换
信号转导
免疫印迹
生物
病理
癌症研究
下调和上调
医学
细胞生物学
生物化学
基因
作者
Bingling Zhang,Feng Wu,Pingping Li,Haiding Li
标识
DOI:10.1080/08923973.2022.2128369
摘要
Our study shows that ARRDC3 could ameliorate CCl4-induced liver fibrosis and EMT progression via the ITGB4/PI3K/Akt signaling pathway, which provides a meaningful reference for the clinical targeted treatment of liver fibrosis.
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