作者
Cong Si Tran,Julia Kersten,Jingyi Yan,Marco Breinig,Thorben Huth,Tanja Poth,Ombretta Colasanti,Tobias Riedl,Suzanne Faure‐Dupuy,Stefan Diehl,Lieven Verhoye,Teng-Feng Li,Marit Lingemann,Philipp Schult,Gustaf Ahlén,Lars Frelin,Florian Kühnel,Florian W. R. Vondran,Kai Breuhahn,Philip Meuleman,Mathias Heikenwälder,Peter Schirmacher,Ralf Bartenschlager,Vibor Laketa,Stephanie Roessler,Darjus F. Tschaharganeh,Matti Sällberg,Volker Lohmann
摘要
High expression of phosphatidylinositol 4-kinase III alpha (PI4KIIIα) correlates with poor survival rates in patients with hepatocellular carcinoma. In addition, hepatitis C virus (HCV) infections activate PI4KIIIα and contribute to hepatocellular carcinoma progression. We aimed at mechanistically understanding the impact of PI4KIIIα on the progression of liver cancer and the potential contribution of HCV in this process.
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