衰老
医学
急性肾损伤
肾脏疾病
癌症研究
肾
下调和上调
纤维化
细胞生物学
病理
生物
内科学
基因
生物化学
作者
Congcong Yao,Wei Wei,Guoyu Wu,Yan Zhang,Keke Sun,Zhiyuan Liu,Yushanjiang Abudureheman,Heng Wu,Qi Lv,Ayinuer Paredong,Songtao Shou,Heng Jin
摘要
DOT1L regulates cellular senescence through the miR-222-5p/WNT9B pathway in AKI. These findings suggest that DOT1L may serve as a potential therapeutic target to mitigate the progression of AKI to chronic kidney disease.
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