Amyloid-β Signal Transduction Antagonist, A Possible Way to Relief Alzheimer’s Disease

作者
Zhengzhe He
出处
期刊: 卷期号:4 (1): 283-286
标识
DOI:10.54254/2753-8818/4/20220567
摘要

As a well-known common neurodegenerative disease in aging, Alzheimer’s disease will cause the loss of the ability to talk, cognization and memorize. Amyloid beta is considered to be a major cause of Alzheimer’s disease. It will form many different types and accumulate in human brain and cause Alzheimer’s disease. However, a recent report about the plagiarism of the fundamental paper of this theory makes many people think that our research direction on Alzheimer’s disease is wrong. The author agrees that the plagiarism of the paper is terrible and should be punished, but the author does not think that this will waste everything we have done about amyloid beta. Amyloid beta is still a major reason that causes Alzheimer’s disease and find a way to stop the accumulation of amyloid beta in brain is a feasible way to cure Alzheimer’s disease. Research on the role of amyloid beta in Alzheimer’s disease, whether it is a major cause is the major purpose of the essay. What’s more a new method that aim to reduce amyloid beta in brain to cure and relief Alzheimer’s disease will be introduced in this article. This method aims to stop the signal transduction of amyloid beta to neuron cells, thus, stop amyloid beta from damaging our brain and cause Alzheimer’s disease. The reason this article is written is to confirm the importance of amyloid beta for Alzheimer’s disease. In addition, introduce a new cure method. Since this method is new, conclude the advantages and disadvantages of this method could inspire other researchers, and improve the performance of the drugs developed based on this method to reach the final goal, which is cure the Alzheimer’s disease.

科研通智能强力驱动
Strongly Powered by AbleSci AI
科研通是完全免费的文献互助平台,具备全网最快的应助速度,最高的求助完成率。 对每一个文献求助,科研通都将尽心尽力,给求助人一个满意的交代。
实时播报
LiJc完成签到,获得积分10
刚刚
不懂发布了新的文献求助10
刚刚
1秒前
四季大枣发布了新的文献求助10
1秒前
星辰大海的应助被lsm采纳,获得10
2秒前
nanami完成签到,获得积分10
2秒前
3秒前
初景发布了新的文献求助30
3秒前
瘦瘦世德完成签到 ,获得积分10
4秒前
4秒前
爆米花的应助被科研通管家采纳,获得10
4秒前
丘比特的应助被科研通管家采纳,获得10
4秒前
干净的琦的应助被min采纳,获得10
5秒前
5秒前
孙波发布了新的文献求助10
5秒前
6秒前
xfya完成签到,获得积分10
6秒前
内向的白玉完成签到 ,获得积分10
8秒前
王宇航发布了新的文献求助10
8秒前
盲点完成签到,获得积分10
8秒前
学麻了完成签到 ,获得积分10
9秒前
鼠鼠完成签到,获得积分10
9秒前
10秒前
11秒前
温暖的台灯完成签到,获得积分10
15秒前
16秒前
嘻嘻完成签到 ,获得积分10
17秒前
还活着完成签到,获得积分10
18秒前
18秒前
晨许沫光完成签到 ,获得积分10
20秒前
清腾完成签到,获得积分10
20秒前
22秒前
22秒前
彩色的舞蹈完成签到,获得积分10
23秒前
溜溜完成签到 ,获得积分10
25秒前
Derek完成签到,获得积分10
25秒前
huakeguanli发布了新的文献求助10
25秒前
26秒前
27秒前
yy发布了新的文献求助10
27秒前
高分求助中
(应助此贴封号)【重要!!请各用户(尤其是新用户)详细阅读】【科研通的精品贴汇总】 10000
Rosenblum, Global Change Biology 800
自動車の空力技術 800
Organizational Behavior 510
Management and the Arts 510
Issues in Task-Based Language Teaching 500
Geschichtliche Grundbegriffe (GGB), Band 5: Pro–Soz 300
热门求助领域 (近24小时)
化学 材料科学 医学 生物 纳米技术 计算机科学 化学工程 工程类 有机化学 物理 复合材料 生物化学 内科学 细胞生物学 基因 遗传学 免疫学 冶金 光电子学 癌症研究
热门帖子
关注 科研通微信公众号,转发送积分 7789717
求助须知:如何正确求助?哪些是违规求助? 9327268
关于积分的说明 20416983
捐赠科研通 7379119
什么是DOI,文献DOI怎么找? 3322867
关于科研通互助平台的介绍 2470788
邀请新用户注册赠送积分活动 2339676