Perioperative Pulmonary Atelectasis: Part II. Clinical Implications.

麻醉 外科
作者
David Lagier,Congli Zeng,Ana Fernandez-Bustamante,Marcos F. Vidal Melo
出处
期刊:Anesthesiology [Lippincott Williams & Wilkins]
标识
DOI:10.1097/aln.0000000000004009
摘要

The development of pulmonary atelectasis is common in the surgical patient. Pulmonary atelectasis can cause various degrees of gas exchange and respiratory mechanics impairment during and after surgery. In its most serious presentations, lung collapse could contribute to postoperative respiratory insufficiency, pneumonia, and worse overall clinical outcomes. A specific risk assessment is critical to allow clinicians to optimally choose the anesthetic technique, prepare appropriate monitoring, adapt the perioperative plan, and ensure the patient's safety. Bedside diagnosis and management have benefited from recent imaging advancements such as lung ultrasound and electrical impedance tomography, and monitoring such as esophageal manometry. Therapeutic management includes a broad range of interventions aimed at promoting lung recruitment. During general anesthesia, these strategies have consistently demonstrated their effectiveness in improving intraoperative oxygenation and respiratory compliance. Yet these same intraoperative strategies may fail to affect additional postoperative pulmonary outcomes. Specific attention to the postoperative period may be key for such outcome impact of lung expansion. Interventions such as noninvasive positive pressure ventilatory support may be beneficial in specific patients at high risk for pulmonary atelectasis (e.g., obese) or those with clinical presentations consistent with lung collapse (e.g., postoperative hypoxemia after abdominal and cardiothoracic surgeries). Preoperative interventions may open new opportunities to minimize perioperative lung collapse and prevent pulmonary complications. Knowledge of pathophysiologic mechanisms of atelectasis and their consequences in the healthy and diseased lung should provide the basis for current practice and help to stratify and match the intensity of selected interventions to clinical conditions.
最长约 10秒,即可获得该文献文件

科研通智能强力驱动
Strongly Powered by AbleSci AI
科研通是完全免费的文献互助平台,具备全网最快的应助速度,最高的求助完成率。 对每一个文献求助,科研通都将尽心尽力,给求助人一个满意的交代。
实时播报
Lucia完成签到 ,获得积分10
刚刚
JAY发布了新的文献求助10
刚刚
涵Allen完成签到,获得积分10
刚刚
Liziqi823完成签到,获得积分10
1秒前
avalin发布了新的文献求助10
1秒前
1秒前
mw完成签到,获得积分10
2秒前
2秒前
asdfqwer的应助被故事的小红花采纳,获得10
2秒前
喝汤发布了新的文献求助10
2秒前
田様的应助被Shimmered采纳,获得10
3秒前
4秒前
4秒前
5秒前
azou完成签到,获得积分10
5秒前
5秒前
万能图书馆的应助被JAY采纳,获得10
5秒前
orixero的应助被认真的不评采纳,获得10
7秒前
yuan1226完成签到 ,获得积分10
7秒前
Zeal完成签到,获得积分10
7秒前
8秒前
藕饼完成签到 ,获得积分10
8秒前
TX发布了新的文献求助10
8秒前
专注白昼发布了新的文献求助10
8秒前
何小抽发布了新的文献求助10
9秒前
9秒前
LZ发布了新的文献求助10
9秒前
无极发布了新的文献求助10
11秒前
Eric_Zhou完成签到,获得积分10
13秒前
Owen的应助被明理诗槐采纳,获得10
13秒前
ad9完成签到 ,获得积分10
13秒前
嘎嘎完成签到 ,获得积分10
13秒前
14秒前
在水一方的应助被SRY采纳,获得10
14秒前
15秒前
15秒前
16秒前
17秒前
17秒前
zzz完成签到 ,获得积分10
18秒前
高分求助中
(应助此贴封号)通过应助OA文献获取积分 10000
The Student's Guide to Social Neuroscience 800
Rosenblum, Global Change Biology 800
Computational Chemical Reaction Engineering: Modeling, Simulation, and Design with MATLAB 600
Organizational Behavior 510
Management and the Arts 510
Production Logging: Theoretical and Interpretive Elements 400
热门求助领域 (近24小时)
化学 材料科学 医学 生物 计算机科学 工程类 纳米技术 内科学 物理 有机化学 化学工程 生物化学 复合材料 光电子学 细胞生物学 心理学 量子力学 催化作用 物理化学 电极
热门帖子
关注 科研通微信公众号,转发送积分 7813322
求助须知:如何正确求助?哪些是违规求助? 9344116
关于积分的说明 20521172
捐赠科研通 7406162
什么是DOI,文献DOI怎么找? 3330418
关于科研通互助平台的介绍 2477074
邀请新用户注册赠送积分活动 2349935